Vaccines Have Serious Side Effects

The Revised Authoritative Guide To Vaccine Legal Exemptions

Comprehensive, authoritative information about vaccine exemptions you can trust, from Alan Phillips, J.D., a leading vaccine rights attorney with years of experience helping clients throughout the U.S. legally avoid vaccines in a wide variety of vaccine-refusal settings. Critical details for parents, students, immigrants, healthcare employees, military personnel and contractors, agencies, attorneys and clientsvirtually anyone concerned with legally avoiding vaccines in the United States. This Guide provides and explains: Important background information about the legal system; How state and federal statutes, regulations, constitutions and legal precedent interact to define the boundaries of your legal exemption rights; How to deal with local authorities and to avoid mistakes that cost others their exemption; Where legal technicalities and practical reality differand what to do about it; Continue reading...

The Revised Authoritative Guide To Vaccine Legal Exemptions Summary

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Contents: 130-page Ebook
Author: Alan Phillips
Official Website: www.vaccinerights.com
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Vaccination Is Not Immunization Vaccine Risks Exposed

Doctor's New E-book Informs Parents About Vaccine Side Effects, The Link To Autism, Swine Flu Scam & More. Ends The Confusion On Whether Or Not To Vaccinate. Many Doctor Testimonials. Here's a look at some of what's covered. . . The disturbing truth: why children are growing sicker, fatter and dumber year after year. The shocking composition of what's really being injected into your child. Think only your children are at risk? Think again. As of 2008, adults are Medicine's newest target, with the Cdc's new Adult Immunization Schedule of 74 vaccines! What is the Germ Theory and why does the medical establishment ignore the errors inherent in it? The implications for your child's health are staggering! Do you know the difference between natural immunity and artificial immunity? 12 deadly reactions to the Dpt vaccine Did you know that the three vaccines contained in this three-in-one injection were never tested together before it was brought to market? In the 1940s. There's more: The truth about autism The evidence is piling up regarding the accine autoimmune brain programming connection and you must not wait another day to get the complete story. The false advertising behind the Mmr (measles-mumps-rubella) vaccine If so many doctors are refusing to give it to their children, what is that telling you? The most amazing statistic put forth from a source with no medical axe to grind Metropolitan Life Insurance. According to the ex-commissioner of the Fda, the number of adverse reactions to vaccines are being woefully underreported. If flu shots worked, why do you and your child get them year after year and why do you and your child keep getting the flu? It's all here. . .plus, the worrisome correlation between flu shots, mercury and Alzheimer's disease. What are toxics in vaccines and why are they being used on your child? And are you sure you want these poisonous substances injected into your child? What mandated vaccination really means and how your child can be made exempt if you choose

Vaccination Is Not Immunization Vaccine Risks Exposed Summary

Contents: Ebook
Author: Tim O Shea
Official Website: www.immunitionltd.com
Price: $25.00

Vaccine Risks Report

This Vaccine Risks Report was compiled from over 50 books, essays, articles and interviews. All of the information is currently available in books, periodicals, and through scientific journals and reports. Many of the statistics, quotes, and detailed information can be found on the Internet now, through websites and search engines. What are Vaccines risks: Do Vaccines have side effects? Are Vaccines Mandatory? What are the facts Im Not being told? Do I have a choice Not to vaccinate? Are there organizations that can help and support my decision? What other kind of science is available? What books or articles should I read? Which websites have extensive details, reports and explanations? How does our government process work?

Vaccine Risks Report Summary

Contents: 23 Page Ebook
Author: Garrett Goldenberg
Official Website: www.vaccinerisksreport.com
Price: $9.99

Other Issues Relating To Infection And Diabetes

Current guidelines from the Centers for Diseases Control (CDC) recommend that all diabetic patients receive influenza and pneumococcal vaccination. Despite these recommendations, a recent review by the CDC and the Council of State and Territorial Epidemiologists (CSTE) suggested that only 52 of diabetic patients reported receiving the influenza vaccination in the past 12 mo and only 33.2 recalled receiving pneumococcal vaccination at all. When prescribing antibiotics in the diabetic patient, particular caution is warranted to avoid nephrotoxicity as well as the potential for eye toxicity. Also, when administering oral antibiotics, the effects of gastropathy on oral absorption should be considered. Maintenance of good hygiene, particularly in the context of foot care, is crucial in patients with diabetes.

Type Diabetes And Possible Association With Viral Infections

It must be borne in mind that many virus variants are grouped together under one name for instance, the term 'Coxsackie virus B4' signifies at least 13 variants. It may be that only a rare variant of the Coxsackie virus B group is diabetogenic and that vaccination against this variant might be possible in the future. Although recent observations suggest that exposures to enterovirus infections both in utero and in childhood may be able to induce and promote fl-cell damage and thus lead to Type 1 diabetes, we do not have enough evidence to say that such infections are truly causing Type 1 diabetes. Many people hope that it will be possible to develop a vaccine to prevent Type 1 diabetes, but much more information is needed before it will be possible to provide sufficient evidence to justify such an approach. Vaccination may not simply remove a disease like Type 1 diabetes. Interestingly, it was noticed that the penetrance of diabetes in female NOD mice reared in a pathogen-free...

Prevention of Type Diabetes

Other therapies may also be tested as researchers learn more about the causes of type 1 diabetes. For example, if auto-immunity to GAD turns out to be a primary trigger of type 1 diabetes, it might be possible to develop a vaccine that would protect people from developing the disease.

Respiratory Tract Infections

Patients with diabetes who develop pneumococcal pneumonia are at an increased risk of bacteremia as well as a higher mortality (25,26). Pneumococcal vaccine response rates are similar to nondiabetic hosts. During epidemics of influenza, there is an increased mortality and an increased incidence of bacterial pneumonia and ketoacidosis in patients with diabetes. The increased incidence of nasal colonization with St. aureus in diabetics combined with reduced pulmonary ciliary clearance in patients with influenza leads to an increased incidence of staphylococcal pneumonia. Influenza and pneumococcal vaccines are therefore recommended for all diabetic patients.

Antimicrobial treatment

Estrogen suppletion in postmenopausal women (oral or vaginal)) 61,60,62 Vaccines (both currently on halt) Urovac 66 Since the adherence of E. coli to the uroepithelial cell is an essential step in the pathogenesis of UTIs, prevention of this would theoretically lead to a decreased incidence of UTIs. Therefore the attention has shifted towards the development of a vaccine, based on the FimH adhesin of type 1 fimbriae of E. coli. In vitro and animal studies have shown that this vaccine can prevent adherence of E. coli to uroepithelial cells and decrease incidence of UTIs in vaccinated monkeys 63,64 . We have demonstrated that addition of vaccine-induced antiserum to uroepithelial cells isolated from diabetic women, also decreases the adherence of type-1 fimbriated E. coli to diabetic uroepithelial cells 65 . At this moment, clinical studies have been discontinued, because although safe, the vaccine proved only 30 effective in young sexually active women. In addition, another vaccine...

Modified LDL Antibodies and LDLContaining Immunocomplexes

The inverse correlations between modified LDL antibody levels and atherosclerosis, together with data obtained in laboratory animals suggesting that modified LDL antibodies are predominantly of the noninflammatory immunoglobin (Ig)M isotype (189) and human studies claiming that IgM antibodies to modified LDL may predominate over IgG antibodies (190) and have a protective effect in relation to the development of atherosclerosis (191) have led to considerable speculation, including the possibility of vaccination against atherosclerosis (192). This seems highly unwarranted, because of several other lines of evidence. First, the proposed protective murine IgM antibodies are predominantly reactive with oxidized phospholipids, although human antibodies reacting with modified lysine groups have been extensively characterized (193,194). Second, when the isotype distribution of modified LDL antibodies has been studied under stringent conditions, using affinity chromatography-purified...

Overview of Diabetes Management Combined Treatment and Therapeutic Additions

IL-2, IL-4, IL-10 and TNF-a Anticytokine and anti-IFN-y antibodies Anti T-cell antibodies, anti-CD3, -CD4, -CD8 Anti T-cell-receptor antibodies Anti-MHC class I and class II antibodies Immunosuppressive drugs (cyclosporine) Immunomodulating agents Adjuvants (BCG vaccine and CFA) Nicotinamide Immunosuppression Therapy. This form of therapy is directed to prevent the action of diabetogenic T cells (in the early stage of insulitis) in order to preserve insulin secretion, at least in the short term. In a study, BCG antituberculosis vaccination was reported to be effective, with a 66 remission in the newly diagnosed treated patients as compared to 7 in the controls. Two other clinical trials with BCG vaccine in newly diagnosed type 1 diabetics have found no beneficial effects.

Prevention Of Type A Diabetes Introduction

In general, the prevention of a disease process requires that certain underlying criteria be met. First, there must be an accurate means of identifying subjects at risk for developing the disease (or an extremely safe intervention such as vaccination) and, second, there must be some intervention that can modulate the disease process. Prediction of type 1 diabetes was discussed in the previous section, and the latter is the focus of this subsequent section. The prevention of type 1 diabetes can theoretically be implemented with three different strategies a primary, secondary, or tertiary approach. The primary prevention strategy involves initiation of an intervention before the onset of autoimmunity. This involves the identification of modulating or precipitating factors in the disease process and intervening in subjects at genetic risk that have no evidence of P-cell damage. The secondary prevention strategy would delay or suppress continued P-cell destruction in subjects with...

Is Disease Prevention Possible

The safest and most rational form of primary prevention would be modification of environmental determinants of disease. Leading candidates in type 1 diabetes are viruses and infant nutrition. The congenital rubella syndrome is sometimes cited as evidence that a virus can cause type 1 diabetes, and that type 1 diabetes can be prevented by routine rubella vaccination. It is, however, far from certain that children with this syndrome have typical type 1 diabetes, and some clearly have an insulin-resistant form of the disease. Associations between enteroviral exposure in utero and subsequent development of diabetes in the child6,7 raise the possibility that women might at some future date be vaccinated against a range of viruses, but the evidence base for such an intervention is far from established. The alternative environmental explanation, that exposure to cows' milk at an early stage of development might be diabetogenic, remains controversial8. The hypothesis is, however, susceptible...

Prevention Of Diabetes

Several trials have demonstrated that immunosup-pressive agents can slow or interrupt the disease process, but concern remains about long-term toxicity. Studies with newer agents (e.g. sirolimus, mycopheno-late, etc.) have been initiated. Early studies with anti-CD3 monoclonal antibodies have suggested a beneficial effect on preservation of p-cell function. Nicotinamide has been suggested as a means of protecting p-cells however, unfortunately a large scale trial (ENDIT the European Nicotinamide Diabetes Intervention Trial) showed no significant benefit. Expansion of p-cell mass (with exendin, GLP-1 or islet neogenesis-associated protein) is a possible research consideration. Rest of p-cells (with insulin therapy) in early-onset diabetes or ICA-positive relatives has not yet shown conclusively that it delays or prevents the development of type 1 diabetes. More recently several groups have made progress towards the development of a vaccine to prevent the onset of type 1 diabetes.

Preventing type diabetes

An example of primary prevention for type 1 diabetes would be vaccination against the viruses that may be associated with diabetes. Unfortunately, doctors haven't yet pinpointed the exact virus or viruses, so vaccinations aren't yet practical. Doctors have tried vaccinations in countries (such as Finland) where type 1 diabetes occurs most often, but the vaccinations didn't stop the number of new cases of type 1 diabetes from rising. Doctors also have considered giving antiviral agents to people who are at high risk of acquiring diabetes, but this approach, although successful, has been tested only in animals so far.

Prevention Strategies

A case-finding strategy, perhaps involving genetic screening at birth, followed by autoanti-body screening in those genetically at risk, followed by appropriate intervention. This case-finding approach is being taken in the DAISY (Diabetes Autoimmunity Study in the Young) (59), DIPP (Diabetes Prediction and Prevention Project) (60), and NOBADIA (Norwegian Babies against Diabetes) (61) studies. In such a strategy, treatment (e.g. vaccination) would be only of susceptible individuals. 2. A population strategy. In this scenario, one might have a treatment approach (e.g. vaccination) sufficiently safe and effective that the entire population could be treated.

Ten of the Latest Discoveries in Type Diabetes

Possibly preventing type 1 diabetes with vaccines and insulin Monitoring blood glucose in new ways Fighting the complications of type 1 diabetes Reversing the loss of beta cells The Development of the GAD Vaccine First he found that patients with T1DM had high levels of GAD autoantibody. Then he discovered that mice prone to develop T1DM could be protected from the disease by receiving small amounts of GAD. He and his colleagues then created a vaccine using GAD that could slow the development of T1DM even after the immune response had begun. The next step was trying the GAD vaccine on humans, which happened in a study in which neither patient nor doctor knew whether the patient was getting GAD vaccine or a placebo. The result was that the GAD vaccine preserved insulin production without any adverse effects. The vaccine is called Diamyd and may prove to be very valuable in protecting high-risk children from developing T1DM. See Chapter 2 for more about GAD.

Where Next

It is logical to believe that improved understanding of disease will point the way to prevention. Happily for us, this is not always necessary. James Lind prevented scurvy with lime juice, Edward Jenner prevented smallpox by vaccination, and John Snow halted a cholera epidemic by removing the handle of the water pump in Broad Street, and all three advances were based on shrewd observation rather than scientific hypothesis.

Viral Infection

Viral infections also may serve as a stress factor, bringing Type 1 diabetes to clinical recognition. In a nationwide incident case control study in Sweden, the odds ratios for Type 1 diabetes for children exposed to 0, 1-2, or over 2 infections during the last year before diagnosis of diabetes revealed a linear increase (OR 1.0, 1.96 and 2.55 for 0, 1-2 and over 2 infections, respectively), which remained significant when standardized for possible confounders such as age and sex of the children, maternal age and education and intake of antibiotics and analgesics (38). This study also found a protective effect of measles vaccination.

Avoiding Sickness

Having diabetes can complicate the management of common illnesses such as the flu, so all people with diabetes should get a flu shot every year. The pneumococcal vaccine is normally given to people over age sixty-five, but if you have diabetes and you are over eighteen years old, you should also get a pneumococcal vaccine to reduce the risk of one type of pneumonia.

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The real value of the effect of any prevention program is not just its ability to prevent type 1 diabetes but also its impact on a range of economic, public health, and ethical issues. Concerns that merit strenuous debate include not just the efficacy of a particular prevention program but also its efficiency, equity, and protection of human subjects. At the moment, with yet incomplete data regarding the efficacy of prevention, several alternatives are being discussed Screening in high-risk populations or screening in the community Screening with genetic or immunologic or metabolic markers Treatment with immunologic vaccines or dietary supplements Each alternative poses different questions regarding the implementation and effect of the prevention program. Several investigators recognize the need to assess the relative costs and benefits (or efficiency) of the programs. It is not clear, though, that equity, accessibility, or privacy issues are included in the requested evaluations....

Epidemiology

A diagnosis of T1D at the age of 30 or above was formerly referred to as latent-onset autoimmune diabetes in adults (LADA). This is not infrequent and it is estimated that about 10 of the population with a pheno-type of type 2 diabetes actually had (autoimmune-mediated) T1D (7). This specific population has anti-GAD or IA-2 antibodies by definition, albeit that in the first 6 months to 6 years after diagnosis these patients do not depend on insulin and that part of them is overweight resulting in concomitant (relative) insulin resistance. In this particular subpopulation, P-cell destruction is only slowly progressive and may take up to 12 years to be complete. An association of the occurrence of T1D with viral infections in early childhood or with vaccination could not be confirmed in recent studies (8).

Tertiary Prevention

A large number of new agents successfully preventing type 1A diabetes in animal models are entering clinical trials. As reviewed by Atkinson and Leiter (98), more than 100 different therapies prevent type 1 diabetes in the NOD mouse model and it is likely that disease prevention may be too easy in this model. The BB rat model is more demanding, but suffers from the presence of a T-cell lymphopenia autosomal recessive mutation that is likely to limit successful immunomodulation. Nevertheless, the existence of these models has stimulated development of agents for type 1A diabetes prevention. Probably the most exciting pathway goes under the rubric of immunologic vaccination. Antigens or peptides of autoantigens can be administered to the NOD mouse and prevent the development of diabetes. The three major molecules studied have been insulin, GAD, and a heat-shock protein. Biotechnology companies are bringing all three molecules to clinical trials, usually in new-onset diabetes patients,...

Primary Prevention

In addition to early infant diet, other environment risk determinants have been implicated in the potential pathogenesis of type 1 diabetes, including enteroviral infections and vaccine administration. These determinants may serve as immune triggers or disease modulators, but, to date, no large trials are focusing on these agents for primary intervention studies (70-75). At present, only congenital rubella infection is clearly associated with risk for type 1A diabetes (76-78).

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