Migraine Headaches Homeopathic Remedies
Headaches can take the joy out of living. They can lower your motivation level so low that you don't even want to do anything about them People with daily headache deserve our deepest sympathy since they must carry on with life's daily duties in spite of it. Often, no medicine helps. Although there are common headaches, sinus headaches, migraines, and others, the causes of all overlap a great deal. Possibly the parasite wormlet comes first, since even young children can suffer with migraines. Strongyloides is a microscopically small threadworm that horses are plagued with, but humans and our pets pick it up easily. Every migraine sufferer (100 ) has high levels of this tiny worm. Perhaps it is really the special bacteria it brings with it that cause the blood vessels to seep or to spasm in the brain, causing pain. Zap it immediately. Is it the toxins made by bacteria or the inflammation from the bacteria or wormlets themselves that produces the headache Certainly, one can eat the...
There is a long list of drugs, mainly centrally acting, that can cause weight gain (Table 10.1), and these are often prescribed to patients with diabetes. Of particular note are tricyclic antidepressants and anticonvulsants such as carbamazepine and gabapentin used for symptom control in painful neuropathy. Other drugs include antipsychotic drugs, notably the newer atypical antipsychotic agents, such as clozapine and olanzapine, that can cause substantial weight gain, and have been suggested to independently worsen insulin resistance and perhaps increase diabetes risk in non-diabetic subjects (Hedenmalm et al., 2002). Pizo-tifen, a serotonin antagonist used in the management of migraine, may cause increased appetite and therefore weight gain (Galanopoulou et al., 1990). Cor-ticosteroids and some progesterone preparations, such as medroxyprogesterone acetate may also cause substantial weight gain, and in the case of corticosteroids, worsen insulin resistance and impair f-cell function....
Preeclampsia is a syndrome of new-onset hypertension and proteinuria after 20 weeks of gestation in a previously normotensive female without proteinuria (Table 1). Women with preeclampsia are often asymptomatic, and the onset may be insidious. Early signs and symptoms may include headache, visual changes, epigastric pain, and edema of the hands and face. More severe complications include renal failure, cerebral edema, cerebral hemorrhage, seizures (eclampsia), pulmonary edema, and HELLP - the syndrome of hemolysis, elevated liver enzymes, and low platelets. Headache, scotomata, epigastric pain
In reality, the problem is by no means as dramatic as that. First, you are quite likely to be woken up by the symptoms offalling blood glucose. You may feel sweaty, restless or irritable. Occasionally, your restlessness may wake your partner even if you remain asleep. It is not unusual to sleep through a severe hypoglycaemic reaction as your body mobilises various hormones in response to the falling level of glucose, which will stimulate the release of stored glucose to correct the situation. After a reaction like this, you will awake with a headache and symptoms much like a bad hangover. Sometimes, there may be a swing too far in the opposite direction, so that your blood glucose rises too far. If you regularly wake up feeling bad with
Where do your fingers go To your mouth to remove a hull or bit of something from your teeth. Or to eat a last bite of something. Or to turn a page of the telephone directory. You have just eaten a culture sampling from your own kitchen sponge. In two hours they are already multiplying in the greatest culture system of all your body You have given yourself your next sore throat, or cold or headache. The worst possible habit is to wipe a child's face and hands with the kitchen cloth. Or to have a handy towel hanging from the refrigerator handle.
Few studies have examined the development of impaired hypoglycaemia awareness during pregnancy although most clinicians would agree that this is particularly problematical during the first trimester. Evers et al. (2002a) observed that severe hypogly-caemia in the first trimester was more likely to occur in women with reduced symptomatic awareness of hypoglycaemia. In laboratory-induced hypoglycaemia Bjorklund et al. (1998a) did measure symptomatic responses to hypoglycaemia during the third trimester, and also postnatally, and found that symptoms such as 'inability to concentrate', 'headache' and 'pounding heart' were less prominent during pregnancy compared with during the postnatal period. However, it is difficult to ascertain whether this is a consequence of differences in glycaemic control or the incidence of hypogly-caemia during these two time periods. Fear of hypoglycaemia is also greater in women who have experienced severe hypoglycaemia (Evers et al., 2002a) and this is an...
Your face may flush and you may feel a surge of adrenaline flowing through your body. Your mouth may become dry and you may feel nauseated. Your breathing may become more rapid and shallow. Your heart may begin to pound and your muscles may become tight, leading to headaches or cramps.
It is the progestin component of hormonal contraceptives that prevents pregnancy. Estrogen is added to decrease intermenstrual bleeding. To prescribe an estrogen-containing contraceptive, the diabetic woman's cardiovascular risks need to be evaluated due to the estrogen-induced changes in her metabolic profile. Estrogen produces a dose-dependent increase in globulin production, increasing coagulation factors and angiotensin II levels, thereby increasing thromboembolic risk and a slight increase in mean arterial blood pressure (9, 25). With low estrogen dose COC (
The most common cranial mononeuropathy is the third cranial nerve palsy. The patient presents with pain in the orbit, or sometimes with a frontal headache (53,71). There is typically ptosis and ophthalmoplegia, although the pupil is usually spared (72,73). Recovery occurs usually over three months. The clinical onset and time-scale for recovery, and the focal nature of the lesions on the third cranial nerve, on postmortem studies suggested an ischaemic etiology (53,74). It is important to exclude any other cause of third cranial nerve palsy (aneurysm or tumour) by computed tomography or MRI scanning, where the diagnosis is in doubt. Fourth, sixth, and seventh cranial nerve palsies have also been described in diabetic subjects, but the association with diabetes is not as strong as that with third cranial nerve palsy.
Hypoglycemia (see Chapter 4) often comes on fast and leaves you with a headache or a general feeling of weakness and sometimes confusion. Because of this, people have believed that low blood glucose, especially if it occurs repeatedly, may destroy mental functioning. People who have had repeated episodes of hypoglycemia have shown no loss of mental functioning. Children may have different results because their brains are still developing.
Primary symptoms of hyperglycemic ketoacidotic decompensation are polyuria, thirst, and loss of weight. Furthermore, these patients complain of weakness, tiredness, headache, lack of appetite, nausea or vomiting. Typical clinical signs of ketoacidosis are the acetonic fetor ex ore and the so-called Kussmaul's respiration. Exsiccosis or hypovolemia manifests clinically as oligo- or anuria (prerenal insufficiency) and hypotension. Further clinical signs of the dehydration are tachycardia, dry tongue, standing skin folds, muscle cramps, and soft bulbi. A special clinical picture is the pseudoperitonitis diabetica, which is a painful tension of the abdominal wall in association with diabetic ketoacidosis. This can be associated with elevated serum-alpha-amylase levels as well as with leukocytosis. It is very important to recognize this clinical picture early and to differentiate it from other causes of an acute abdomen, to avoid unnecessary surgery.
Controlled trial (systolic hypertension in Europe trial investigators) clearly showed an impressive CV morbidity and total mortality reduction in the group that received nitrendipine alone 14 . Another large study, the Hypertension Optimal Treatment (HOT), demonstrated that the intensive control of BP, reduced the CV events more significantly than in the group whose BP was less tightly controlled 52 . The beneficial effects were of significantly greater magnitude in the diabetic cohort however, many of the diabetic patients also received ACE inhibitors. Other recent studies, however, such as the Fosinopril Amlodipine Event Trial (FACET) 53 and the Appropriate Blood Pressure Control in Diabetes (ABCD) 54 trials, showed that compared with ACE inhibitors, treatment with dihydropyridine CCBs in diabetic hypertensive patients might be harmful. This raised significant concern about the safety of dihydropyridine CCBs in these patients. Overall, CCBs are well-tolerated the major side effects...
Brandi Rainey, age 34, of Amish religious culture, was diagnosed with MS four months earlier after an MRI confirmed it although she had symptoms for many years. She was told she had inherited a gene for it and that Amish folk are particularly susceptible to MS for reasons of inbreeding. She had a constant pain running down the side of her neck, and headache. Her legs were getting too heavy to get up stairs. Our tests showed her brain was full of scandium (tooth metal alloy) and fluoride (toothpaste). Her vision was getting worse her eyes were full of wood alcohol. She lost no time in getting dentures there were no teeth that could be saved. She had several bacteria growing in her jaw bone Strep G (sore throat bacteria), Staphylococcus aureus (this was raising her pulse to over 100), Clostridium tetani (causes great stiffness), and Shigella (produces nerve toxins). She killed these with a frequency generator. Five weeks later the pain and stiffness in her neck were gone, her pulse was...
I Severe hypoglycemia, which is marked by a blood glucose of less than 55 mg dl, requires the help of someone else to restore the patient's blood glucose. Someone with severe hypoglycemia may be unconscious. It can leave the person with a severe headache and unable to function for lack of glucose in the brain. A shot of glucagon is in order in this situation.
Patients with mitochondrial DNA (mtDNA) disease may have a clinical syndrome that involves mitochondrial dysfunction that is easy to identify. As an example, patients with mitochondrial encephalopathy with lactic acidosis and stroke-like episodes (MELAS) present in childhood with short stature and develop bilateral deafness in their teens, and then diabetes, seizures, stroke-like episodes, and an encephalopathy in their third or fourth decade (14). Other patients have a constellation of clinical features that are highly suggestive of mitochondrial disease, but do not fall neatly into a specific syndrome category. An example is a history of migraine and diabetes. Finally, mito-chondrial diseases may have a non-neurological presentation, such as diabetes alone. Between 0.5 and 1 of adult diabetic patients are thought to harbor a causative mtDNA mutation (15).
Cerebral edema typically presents 4-12 h after the treatment for DKA is started (28, 29), but can occur at any time. In up to 5 of cases, cerebral edema may present prior to initiation of DKA therapy (6, 28-30). Signs and symptoms are variable but include headache, mental status deterioration, inappropriately slowed heart rate, and increases in blood pressure (31, 32). Subclinical presentation of cerebral edema is more common (33, 34) and may be associated with smaller third and lateral ventricles during therapy (33). Glaser and colleagues measured intercaudate widths of the frontal horns of lateral ventricles in 41 children and found narrowing in 54 just after initiation of DKA therapy as compared to after recovery (27). Children with narrowing were more likely to have Glasgow Coma Scale (GCS) scores below 15 during therapy (p 0.03), but did not exhibit neurological abnormalities sufficient for diagnosis of symptomatic cerebral edema.
Immune intervention with human intravenous immunoglobulin (IVIg) has become appropriate in some patients with forms of peripheral diabetic neuropathy that are associated with signs of antineuronal autoimmunity (94,95). Chronic inflammatory demyelinating polyneuropathy associated with diabetes is particularly responsive to IVIg infusion. Treatment with immunoglobulin is well tolerated and is considered safe, especially with respect to viral transmission (96). The major toxicity of IVIg has been an anaphylactic reaction, but the frequency of these reactions is now low and confined mainly to patients with immunoglobulin (usually IgA) deficiency. Patients may experience severe headache because of aseptic meningitis, which resolves spontaneously. In some instances, it may be necessary to combine treatment with prednisone and or azathioprine. Relapses may occur requiring repeated courses of therapy.
Were speculative and based only on limited animal research. In contrast, clinical experiences with humans indicated that glucosamine supplements had a tendency to slightly lower blood sugar levels, which would reduce the risk of diabetes. One researcher reported that glucosamine supplements improved wound healing, reduced headaches, and eased inflammatory bowel disease in patients. None of these side benefits were reported by the above-named Berkeley and Tufts publications.
This rare condition is an acute disease that affects successively the air cavities of the face, the orbit, and the brain, in relation to proliferation of a fungus of the class Phycomyceta (56). In 36 of cases it is associated with diabetes, especially in patients with diabetes with ketoacidosis. After an episode of rhinological involvement with epistaxis, a patient with diabetes in acidosis manifests violent headaches and orbitonasal pains with swelling of the lids and ophthalmoplegia. The disease spreads to the meninges and to the brain through the arteries, inducing thrombosis of the ophthalmic then of the internal carotid artery with subsequent hemiplegia. The prognosis is extremely poor.
The aim is to induce regression of new vessels and sometimes to seal leaking new vessels. It is also used to treat maculopathy. Laser treatment prevents severe visual impairment in the majority of patients although the results for maculopathy are less predictable because treatment is close to the macula. Patients should understand that laser treatment may not improve vision but it should stop major deterioration. The treatment is usually given in one or more 30-60 minute sessions as an out-patient. Local anaesthetic and dilating eye drops are used and the patient just has to remain still and concentrate while the treatment is given. Afterwards there is blurring of vision, photophobia, and sometimes eye discomfort or headache. Patients who complain of severe pain should be referred to the eye casualty service.
Are generated as a result of brain neuronal glucose deprivation (3-7,10-12). The brain is vulnerable to any glucose deprivation and neuroglycopenia causes a rapid impairment of cerebral function. These symptoms include abnormal mentation, irritability, confusion, difficulty speaking, ataxia, paresthesias, headaches, stupor, and eventually (if untreated) seizures, coma, and even death (3,7-10,20-23). Neuroglycopenic symptoms can also include a wide array of transient and at times idiosyncratic focal neurological deficits (e.g., diplopia, hemiparesis) (3,7,20) (Table 2).
Her asthma was aggravated by the sinus problems, and Georgia found herself taking increasingly stronger medications. She also was having frequent and severe headaches. At age fifty-two, she consulted holistic physician Robert S. Ivker, D.O., of Denver, Colorado, author of several books, including Sinus Survival, Asthma Survival, and Headache Survival.
Over two years Anita had gained thirty pounds and was now experiencing frequent headaches and suffering joint pain in her hips, shoulders, and hands. Her medical doctor prescribed ibuprofen, and a rheumatolo- Anita's response was dramatic. After three weeks she had lost ten pounds and her glucose had normalized, enabling her to stop taking the glucose-lowering medications. Her pain, swelling, and stiffness decreased considerably, and her energy levels began increasing. At a six-week follow-up visit Anita had lost a total of eighteen pounds, and her blood pressure was normal, so she was able to stop taking the hypertensive medications. In addition, her joint pain was almost entirely gone, flaring up only when she went off her diet of simple, wholesome foods. Anita no longer had a need to take cortisone drugs for her lupuslike symptoms. Her headaches were gone, her energy levels were better, and she actually looked younger.
Many of my patients have claimed that they can tell their blood glucose level by how they feel, and I have challenged them to prove it. Guess who wins every time with the exception of significant hypoglycemia Sure, if your blood glucose is below 50 mg dl and you are sweaty and have palpitations and a headache, you know that you are low but even then, you don't know how low. Therefore, you don't know how much treatment to give yourself to bring it back up, but not too high.
Although discussed in detail in Chapter 22, a brief description of those associated with painful symptomatology is provided here (7,8). Those cranial mononeuropathies affecting the nerves supplying the external ocular muscles typically present with sudden onset of diplopia and an ipsilateral headache often described as a dull pain coming from behind the eye. Similarly, many of the focal limb neuropathies including entrapment neuropathies (7) might present with painful symptoms in the area supplied by the individual nerve. The tarsal tunnel syndrome, which is analogous to the carpal tunnel syndrome in the upper limbs, may present with localized foot pain, which should be distinguished from the pain of the diffuse sensorimotor neuropathy.
Excessive intake of vitamin D in fortified food, over-the-counter supplements or excessive ingestion of anti-rickets pharmaceuticals can result in vitamin D poisoning. An acute toxic dose has not been established but the chronic toxic dose is more than 5O OOO IU day in adults for 1-4 months and, in children, 4OO IU day is potentially toxic. Acute toxicity effects may include muscle weakness, apathy, headache, anorexia, nausea, vomiting, and bone pain. Chronic toxicity effects include the above symptoms and constipation, anorexia, polydipsia, polyuria, backache, hyperlipidemia, and hypercalcemia. Hypercalcemia may cause permanent damage to the kidney (see http www.emedicine.com emerg topic638.htm). Arterial hypertension and aortic valvular stenosis can also result from hypervitaminosis D.
In the presence of DKA, confusion and headache may occur. Patients with new or pre-existing T1DM and acute ketoacidosis may have abnormal electroencephalograms (EEG). In a study of 39 patients aged from 11 months to 16 years who underwent serial EEGs at 1, 12, 24 h, and 5 days after initiation of treatment for DKA, 30 patients were found to have abnormal studies initially (23). EEG severity correlated with serum glucose, osmolality, bicarbonate, -hydroxybutyrate and acetoacetate levels, but did not correlate with pH and glycosylated hemoglobin. Abnormal EEG findings were resolved in 20 of 30 patients in 5 days. EEGs were repeated 2-5 months after treatment five of seven patients undergoing repeat EEG had persistent abnormalities despite DKA resolution.
At that time, we can theorize that a new large parasite was making its appearance. Was a new animal association taking place in the early 1900's Life was indeed becoming more urban with horse manure on all streets. Owning a dog for a house pet was becoming an acceptable lifestyle. Could the tapeworms of these animals give us a tapeworm stage that hosts polio virus Many polio sufferers also had migraines. These are caused by Strongyloides, a horse threadworm. Does Strongyloides host polio virus
This variety of depression is associated with Strongyloides, as the main parasite in the brain. Plus chlorine as an allergen Strongyloides is the same worm that causes migraines and other severe types of recurrent headache. It probably depends on where these tiny worms have set up their housekeeping which symptoms you get manic depression or migraines. Maybe chlorine is the deciding factor. Lena Constantine, age 39, had a history of migraine, TMJ, heavy clotting with periods and numerous pains but it shouldn't have made her try suicide 1 years ago. She was put on Prozac afterward. She was parasitized by intestinal flukes (in the intestine), dog whipworm, Strongyloides and human liver flukes. She was started on a parasite program and kidney cleanse. This made her feel so good she took herself off Prozac and landed in the hospital for reasons she couldn't remember. After 42 days spent there she got out, wiser than before. She set to work again, leaving no detail undone, because she...
Many people with diabetes take oral medications along with insulin or alone to better manage their condition. These medications come in various categories, based on type, and each works differently. They can have side effects, including upset stomach, low blood glucose, weight gain, liver failure, headache, and fluid retention. They must be taken as prescribed and under a doctor's care. Studies have shown that diabetes and its complications might be prevented by these oral medications. This is especially true when the person taking them also eats a healthy diet and gets adequate regular exercise.
Last night, she barely slept because of her stuffy nose and headache. She decided she needed a liquid nighttime cold medicine so she wouldn't have to spend another night like that. At the drugstore, there were so many choices, Evelyn decided to ask the pharmacist to help her choose which medicine would work best for her. Her blood glucose was already running high because of her cold, and she didn't want to raise it any higher.
Michelle Whorton had stomach pain at the middle of her abdomen, not related to eating. She had occasional very bad diarrhea and also daily headaches. She was started on the kidney herbs for other reasons. Her diarrhea disappeared We found she had Ascaris (probably in her stomach where they cause indigestion and inflammation). She zapped them with a frequency generator set to 408 KHz for three minutes at 10 volts. She was to be very careful with sanitation since they owned a number of farm dogs. Next seen after six weeks, she stated that all her previous problems were gone but she had a different pain in the mid-lower abdomen that got worse during her period and sent pain shooting down both legs.
Patients with ischemic stroke usually present with focal neurological deficit of sudden onset. Occasionally a more gradually or stepwise onset can be found, especially in patients with a hemodynamic origin of cerebral ischemia. Common deficits include dysphasia, dysarthria, hemianopia, weakness, ataxia, sensory loss, and cognitive disorders such as spatial neglect (9). Symptoms are unilateral except in some patients with posterior circulation stroke. Most patients remain alert. Mild-to-moderate headache is an accompanying symptom in about a quarter of all patients with ischemic stroke and is more common in stroke of the posterior circulation. Patients with a dissection of the carotid, vertebral, or intracranial arteries usually complain of severe headache in the early course of their illness, sometimes even for days preceding the actual stroke. By definition, signs and symptoms of stroke last for more than 24 h deficits of shorter duration that are caused by focal cerebral ischemia...
Topiramate is a fructose analog that was initially examined because of its antidiabetic possibilities. Although it is an anticonvulsant used in complex partial seizures, topira-mate was recently shown to be efficacious in the management of neuropathic pain (116). Unfortunately, it was first examined only in normal animals and had no hypo-glycemic properties. It has now undergone extensive testing for epilepsy, migraine, involuntary movements, central nervous system injury, and neuropathic pain. The first two studies used a titration to 400 mg day, which was associated with fairly severe central nervous system side effects, which were prohibitive. The studies failed to establish an effect in diabetic neuropathic pain. A third study using different end points, with
Exenatide application in humans leads to antibody formation against this lizard peptide in more than 30 of the cases after prolonged administration. However, this has not yet been associated with any reduction in efficacy, nor do those antibodies appear to cross-react with native human GLP-1. Adverse reactions after subcutaneous administration include nausea and vomiting, and sometimes headache, in a dose-dependent manner. Nausea is transient and most pronounced at the beginning of exenatide treatment. For this reason, a dose titration from 5 (xg twice daily to 10 g twice daily after 4 weeks is recommended when starting therapy. The recommended dose for long-term treatment is 10 (xg twice daily before breakfast and before dinner. Hypoglycemia has not been reported during monotherapy, but can occur when exenatide is administered in combination with sulfonylureas 15 .
Cerebral edema begins with headache followed by neurological deficits (37). Early signs are severe headache, incontinence or lowered mental status. In this situation, an early therapy with hypertonic manitol solution as bolus i.v. in a dose of 0.5 to 2.0g kg body weight can be helpful (37). Manitol is an osmotic active plasma expander which reduces blood viscosity and leads to an elevation of peripheral blood flow to induce an increase of cerebral oxygenation. This effect is observed within 15 to 30 minutes after injection of the bolus and can be observed then for approximately 6 hours. Control of plasma osmolality and creatinine are needed. The administration of dexamethasone and hyperventilation are of no evident benefit (38).
A condition that often precedes type 2 diabetes, or may be a consequence of insulin resistance. In older adults, correcting magnesium depletion may improve insulin response and action. Several clinical studies have reported that magnesium supplementation is beneficial in reducing blood glucose levels, improving insulin control, and stabilizing the metabolic syndrome cascade 20, 21 . Low magnesium intake is also associated with an increase in inflammatory markers, such as C-reactive protein and E-selectin. Symptoms of low magnesium status can include restless legs, muscle tightness and cramps, palpitations, arrhythmias, headaches, muscle weakness, and fatigue. Drugs that deplete magnesium include hyrochlorthiazide, oral contraceptives, and corticosteroids. The major dietary sources of magnesium intake include whole grains, legumes, nuts, and green leafy vegetables.
Dolores Bollapragada, 50ish, had suffered from clinical ergot poisoning in the past which put her in a coma for several weeks. Ergot is a grain (especially rye) fungus, very toxic to the liver. Its byproducts are used in migraine medicine. She had overdosed, somehow. Perhaps her liver never recovered. It no longer detoxified solvents for her, allowing them to accumulate in her retina. There she had propyl alcohol, benzene, carbon tetrachloride acetone, butyl nitrite, styrene, gasoline, wood alcohol, paradichlorobenzene (moth balls), pentane, methylene chloride and decane. She stopped snacking on cold cereal through the day. She had many habits and products to change. But she was determined to salvage her sight. She did.
Understood, but may relate to overly aggressive therapy, the use of hypotonic replacement fluids, local cerebral overhydration and abnormalities of vasogenic function 19 . Symptoms frequently develop 4-12 h after initiation of therapy and include headache, altered mental status, specific neurological deficits and signs of increased intracranial pressure. Treatment with mannitol or hypertonic saline may be beneficial.
Angiotensin II system by diuretics can be counteracted by ACE inhibitors or ARBs. Dose-dependent ankle edema from CCBs is less frequent with the addition of diuretics 78 or ACE inhibitors 55 because of the lower dosage requirements of CCBs. The adverse effects of CCBs (eg, headache, flushing, ankle edema) and ACE inhibitors (eg, cough) are less frequent when low dosages of CCBs were combined with small dosages of ACE inhibitor therapy 55,79 .
Obstructive sleep apnoea may affect as many as 40 per cent of these children (Mallory et al., 1989 de la Eva et al., 2002). Snoring is anon-specific symptom but those children with a history of sleep apnoea, burning headaches or excessive daytime sleepiness should be referred for further evaluation (Gaultier, 1995).
Recommended blood glucose levels are different for children and adults, because of children's high risk and vulnerability to hypoglycemia, relatively low risk of complications before puberty, and developmental and psychological issues. The blood glucose levels at which hypoglycemia is treated are also often higher than the standard recommendations for adults. Any time your child's blood glucose level falls below the value you have established, he or she may have hypo-glycemia. Signs of hypoglycemia include nervousness, shaki-ness, sweating, irritability, impatience, chills, clamminess, rapid heartbeat, anxiety, light-headedness, and hunger. When hypo-glycemia begins to affect the brain, your child may also appear sleepy, angry, uncoordinated, or sad. She may also experience nausea, blurred vision, tingling or numbness in the lips or tongue, nightmares, crying out during sleep, headaches, or strange behavior. In severe stages, confusion, delirium, personality changes, and...
101 Power Tips For Preventing and Treating Headaches
Are you fed up with your frequent headache pain? 101 Simple Ways to Attack Your Headache BEFORE the Pain Starts Guaranteed No Pain, No Fear, Full Control Normal Life Again Headaches can stop you from doing all the things you love. Seeing friends, playing with the kids... even trying to watch your favorite television shows.