Cure Eye Floaters Naturally

Eye Floaters No More

Daniel Brown created Eye Floaters No More Program with the main aim of helping people get lid of those stressful and annoying eye floaters. Daniel is a professional medical researcher and was once affected by these annoying and stressful eye floaters. Because of this, he spent a significant amount of his time researching and writing about their natural cure. The program is packed with a lot of interesting and helpful information that will help you along the way. The book also features some of the best bonuses that you can get immediately you purchase it which includes Vision without glasses and Stress no more. Eye floaters no more is a very helpful eBook for those people that wants to get rid of the annoying eye floaters at the comfort of their homes. You will at the same time get bonuses that will help you deal with and manage stress. Upon purchase, you will get a complete program in form of an eBook. You will also get another two bonuses in form of downloadable eBook. This program contains information that can help people deal with eye floaters regardless of their age, social status and race. Read more...

Eye Floaters No More Summary


4.9 stars out of 29 votes

Contents: EBook
Author: Daniel Brown
Official Website:
Price: $37.00

Access Now

My Eye Floaters No More Review

Highly Recommended

Of all books related to the topic, I love reading this e-book because of its well-planned flow of content. Even a beginner like me can easily gain huge amount of knowledge in a short period.

All the modules inside this e-book are very detailed and explanatory, there is nothing as comprehensive as this guide.

What are the risks of exercise in diabetic persons

Despite the beneficial effects that were mentioned above, physical activity may also be accompanied by unwelcome effects and incur risks for the exercising person, especially if certain conditions and safety rules are not fulfilled. These risks (apart from the already mentioned hypoglycaemia), are sudden death, serious arrhythmias, myocardial infarction etc., especially in those with underlying heart disease (very common in diabetic persons). In diabetic patients specifically, certain diabetic complications can restrict physical activity levels or even preclude some forms of muscular activity. For example, patients with neuropathy should avoid long-term walking and exercises that entail jumping (risk of musculoskeletal trauma) those with nephropathy should be advised to avoid intense exercises (risk of increase in proteinuria) and persons with proliferative retinopathy should avoid weightlifting and very heavy exercises with a chance of hurting themselves (risk of intraocular...

Problems of Establishing the Cause of Death at Postmortem

The measurement of the glucose concentration in the vitreous humour presents similar problems because of continued post-mortem glucose utilisation, and so this cannot be used to confirm ante-mortem hypoglycaemia (false positive). A normal or raised glucose concentration in the vitreous humour after death, however, excludes hypoglycaemia at the time of death (true negative). Thus, the sensitivities and specificities of blood and vitreous humour measurements of glucose in diagnosing ante-mortem hypoglycaemia are unknown.

Fluorescein angiography of the fundus

Hyperfluoresence is the term used whenever fluorescence is increased in an area either due to leakage, as in macular oedema, or due to a window defect that allows the choroidal fluorescence to show through an atrophic retina, as in macular degeneration. It can show as an area of pooling, as in pigment epithelial detachment (Rabb et al. 1978 Gass 1997).

Screening for Complications at Diagnosis

Direct ophthalmoscopy should start with the lens at zero and a red reflex obtained. When present, this indicates that there is no significant evidence of a cataract, vitreous haemorrhage, or retinal detachment. By setting the lens at +10 initially, and using a succession of less powerful lenses, a direct inspection of the cornea, anterior chamber and lens is possible. Diabetic retinopathy should be looked for after pupillary dilation using 0.5-1.0 tropicamide eye-drops. Relative contraindications for this include those with previous eye surgery, lens implants, or history of narrow-angle glaucoma. The precipitation of previously un-diagnosed acute glaucoma, although distressing at the time, may be a service to the patient in the long run since treatment may prevent further visual loss. Patients with scattered microaneurysms and blot haemorrhages require review at 6 months. Diabetic maculopathy can be sight-threatening and requires urgent referral to the ophthalmologist. Other reasons...

Laser Treatment Of Diabetic Retinopathy

There are several possible complications of panretinal photocoagulation. First, as the abnormal, new retinal vessels involute they may bleed, giving rise to a vitreous haemorrhage which will reduce vision for the patient and prevent further assessment of retino-pathy until it clears. Usually resolution of the haemorrhage occurs spontaneously over some months. If the haemorrhage is slow to resolve or retinal assessment felt essential, an operation to remove the vitreous and clear the haemorrhage may be carried out (vi-trectomy). The timing of this is judged on the individual case. Especially in the elderly, the neovascular process can be slow and vitrectomy carries the risk of introducing infection to the eye as well as causing retinal detachment.

Dealing with Eye Problems

Elderly people with diabetes are at risk for the eye problems brought on by the disease, and these problems can affect all aspects of proper diabetes care. Older patients often get cataracts, macular degeneration, and open angle glaucoma in addition to diabetic retinopathy (see Chapter 5).

Treatment Of Established Disease

Anecdotal reports suggest intraocular injection of triamcinolone acetate into the vitreous is helpful in eyes with macular edema however, there are no randomized clinical trials proving its efficacy in diabetic patients (25,31,32). In addition, it has been associated with elevations in intraocular pressure leading to glaucoma, cataract formation, endophthalmi-tis, vitreous hemorrhage, and retinal detachment (33,34). Despite these risks, the anecdotal evidence of its efficacy has made it a valuable option for the treatment of refractory macular edema. Laser therapy, unfortunately, is at times unable to deter the progression of neovascularization and secondary vitreous hemorrhage and traction retinal detachment. In these cases, the risk of severe visual disability rises dramatically and surgical intervention is necessary with vitrectomy. The timing of vitrectomy is important as found in the Diabetic Retinopathy Vitrectomy Study (DRVS). The DRVS enrolled 370 eyes with advanced, active...

Proliferative retinopathy

Figure 5-2C shows an eye that suffers from proliferative retinopathy. If untreated, this condition results in partial or complete loss of vision. Just as in many other parts of the body, when the blood supply in the eye is reduced, new vessels form to carry more blood to the retina. When this happens, the patient is entering the stage of proliferative retinopathy. The blood vessels grow into the vitreous body where they can hemorrhage and block vision. A hemorrhage forms a clot, which contracts, pulling up the retina to produce retinal detachment. The lens can no longer focus the light on the macula, resulting in complete loss of vision. If a person with T1DM has already experienced retinal detachment, an operation called a vitrectomy is necessary. Under general anesthesia, the vitreous body is replaced with a sterile solution. Attachments to the retina are cut, and the retina falls back into place. Vitrectomy restores vision 80 to 90 percent of the time.

Patient Interview

Occurred during a lecture and produced a 'thick mist' in front of his eye within 15 minutes, an operation was necessary. This was not a success and Marcus became blind in his right eye. The other eye was also suffering. Despite laser treatment and removal of the vitreous humour, the doctors at a specialist clinic could not save it and Marcus now has only 3 vision in this eye. 'That's enough, in good light, to see someone opposite me as a dark shape.'


Data from cross-sectional studies indicate that older diabetic patients (middle-age onset) have greater rates of retinopathy and neuropathy than younger diabetics (5), which is likely due to longer duration of disease and greater glycemic exposure. Screening and treatment of these conditions are similar in older and younger patients, but with special considerations such as the potential for adverse effects of drug therapy e.g., angiotensin converting enzymeinhibitors (ACE-I) and drug-drug interactions. Elderly patients with compromised renal function should be evaluated for other renal abnormalities, including genitourinary tract obstructions and infections, which may be common in this population. Older patients with diabetes are also at increased risk of vision loss as a result of glaucoma, cataracts and macular degeneration, so regular examination by an ophthalmologist is essential. Routine podiatric care is also important, since impaired vision and mobility and concomitant...

Diabetic Retinopathy

The importance of the recognition of preprolifera-tive retinopathy is that it indicates the need for urgent referral to an ophthalmologist. New vessels originate from a major vein (occasionally from arteries) and appear in the retinal periphery or on the optic disc. They are much less common in type 2 diabetes than in type 1 diabetes. New vessels have a devastating impact on vision when they burst and produce sudden pre-retinal or vitreous hemorrhage. Contraction of associated fibroglial tissue may result in retinal detachment with resultant loss of vision, which may be profound if it affects the macula. Photocoagulation may also be used for the treatment of macular edema, with focal treatment given for discrete lesions and diffuse treatment for widespread capillary leakage and non-perfusion. Vitreoretinal surgery may be performed to treat severe vitreous hemorrhage and retinal detachment.

The Snellen chart

Snellen Chart Hcvd

Number of letters, as compared with earlier charts with only one or two letters for the 6 60 and 6 36 visual acuity lines, and a large number of letters for the 6 6 and smaller visual acuity lines. Third, the spacing between letters is proportional to the letter size, while the older acuity charts had unequal spacing between letters. Finally, the change in visual acuity from one line to another is in equal logarithmic steps, where there were very small changes for different lines at the small-letter end and rather large changes for the big-letter end of the older charts. This new eye chart permits more precise definition of visual acuity, especially at levels of diminished visual acuity. It is thus used in the current UK national study of photodynamic therapy in age related macular degeneration (TAP study group 1999 Bames et al. 2004). In addition, new methods of scoring responses to this type of visual acuity chart can provide greater sensitivity and reliability of measure. The ETDRS...


Traction retinal detachment (elevation of part of the retina associated with fibrous tissue) persistent hemorrhage, for active retinopathy requiring laser (which is prohibited by hemorrhage) or for retinal detachment that threatens the macula Every 1-3 months Vitrectomy if threatening macula otherwise careful observation The goal of managing PDR is to prevent vitreous hemorrhage and retinal detachments. Pan-retinal photocoagulation (PRP) involves the application of approximately 1500 to 2000, or more, laser burns to the peripheral retina, effectively ablating large areas of ischemic retina. It is believed that this, in turn, reduces the production of angiogenic substances, such as VEGF. Vitrectomy, the surgical evacuation of the vitreous cavity, plays a vital role in the management of severe complications of diabetic retinopathy. Removal of the vitreous limits the progression of neovascularization, because it eliminates the collagen fibrils that act as scaffolding upon which new blood...

Some More Factors

Smoking is a factor that one would assume to be a real problem when it comes to exacerbating retinopathy. However, there is not as strong an association as one would think. The literature goes back and forth on the issue, and it is therefore not fully justified to include smoking with the above pantheon of clear-cut risk factors.7 However, smoking clearly worsens other problems, such as large-vessel disease and renal failure, and these in turn can exacerbate retinopathy. Besides, smoking can aggravate other ophthalmic problems such as cataracts and macular degeneration, so you should feel free to nag patients about their smoking, whether they have diabetes or not. The American Academy of Ophthalmology makes a nice handout on the subject of smoking and its effects on the eye. Unfortunately, large corporations have worked very hard to make it very difficult to stop smoking, so it is not clear how much success you may have with this one. Sometimes, though, patients will be more worried...


The occurrence of large areas of capillary occlusion heralds the onset of PDR. Retinal ischemia occurs in other retinovascular diseases other than diabetic retinopathy, including branch vein occlusion, central vein occlusion, retinopathy of prematurity, and several others. These diseases are collectively called ischemic retinopathies. Retinal ischemia causes increased levels of hypoxia-inducible factor-1 (HIF-1) in the retina (15) and increased expression of genes that contain a HIF-1-binding site in their promoter region, including vascular endothelial growth factor (VEGF) and VEGF recep-tor-1 (16-18). Increased VEGF signaling plays a central role in the development of retinal neovascularization (for review, see ref. 19). Retinal neovascularization grows through the internal limiting membrane (ILM) of the retina onto the surface of the retina and into the vitreous. The new blood vessels leak and bleed resulting in vitreous hemorrhage. Glial cells and retinal pigmented epithelial...


Stress testing is imperative before embarking on an exercise program. Blood pressure should be controlled and guided by the response to exercise testing. Self-monitoring of blood glucose is particularly important in patients taking insulin. Although exercise does not normally aggravate diabetic neuropathy and may even reduce or delay the risk of ophthalmic complications, straining, as seen in heavy resistance training, should be avoided by those with proliferative retinopathy because of the increased risk of vitreous hemorrhage and retinal detachment. It is not known whether patients who have undergone laser procedures can tolerate more aggressive resistance activity (13).

Odds Ends

Traditionally, any patient with a sudden change in floaters needs same-day service to rule out a retinal tear. Because diabetics may have off-and-on hemorrhaging for years, and because such symptoms rarely indicate an acute problem like a retinal tear or detachment, there is a tendency to bend this rule. In an ideal world, it would be best to get everyone in quickly just to be safe. It will relieve the patient's anxiety, and you may occasionally have a chance to get some laser in before a hemorrhage spreads around, or you may rarely find something unexpected like a retinal tear. Practically speaking, however, if you have a lot of diabetic patients you could bring your practice to a standstill trying to get everyone in immediately something that is usually not necessary for patients who have already had occasional hemorrhages and who have longstanding disease that is otherwise well controlled. You will even find that diabetics who have intermittent hemorrhages will ask you if it is...

Eye Floaters No More Official Download Page

The best part is you do not have to wait for Eye Floaters No More to come in the mail, or drive to a store to get it. You can download it to your computer right now for only $27.00.

Download Now